+ Resolve Article
+ Follow Us
Follow on FacebookFollow on Facebook
Follow on TwitterFollow on Twitter

+ Translate
+ Subscribe to Site Feed
GeoScience Most Shared ContentMost Shared Content

Talo-calcaneal relationship in clubfoot

, : Talo-calcaneal relationship in clubfoot. Journal of Pediatric Orthopedics 21(1): 60-64

Talo-calcaneal angles were measured on anteroposterior, stress dorsiflexion, and plantarflexion lateral radiographs of 75 normal feet and 145 clubfeet. The talus and calcaneum from 15 normal fetal limbs were dissected without disturbing the subtalar capsule. Anteroposterior and lateral radiographs of these specimens were also obtained. The long axes of the ossific nuclei and the long axes of the cartilaginous anlagen of the bones were marked, and the talo-calcaneal angles were measured. The talo-calcaneal angles were lower in clubfeet than in normal feet, but there was considerable overlap in the ranges of normal and clubfeet for all the angles measured. The lateral talo-calcaneal angles in normal feet were higher in dorsiflexion than in plantarflexion, whereas the converse was true in clubfeet. The talo-calcaneal angles measured from the axes of the ossific nuclei of the fetal specimens were higher than those measured from the axes of the cartilaginous anlagen. Using logistic regression analysis, a mathematical model was made to predict the probability of correction of clubfeet. A lateral talo-calcaneal angle difference (between the stress dorsiflexion and plantarflexion angles) of 20 degrees suggests that there is a 93% probability that the hindfoot deformity of clubfoot has been adequately corrected. A talo-calcaneal angle of 30 degrees or a talo-calcaneal index of 40 degrees does not ensure correction of clubfoot. A decrease of the talo-calcaneal angle by up to 10 degrees occurs as the child grows because of the alteration of the shape of the ossific nucleus of the talus that occurs normally with growth.


PMID: 11176355

Other references

Deahl, S.T.; Ruprecht, A.; Gilbaugh, G., 1991: Management of submandibular gland sialoliths. Iowa Dental Journal 77(2): 13, 38-13, 38

Dean, F.B.; Hurwitz, J., 1991: Simian virus 40 large T antigen untwists DNA at the origin of DNA replication. Simian virus 40 large tumor antigen (SV40 T antigen) untwists DNA at the SV40 replication origin. In the presence of ATP, T antigen shifted the average linking number of an SV40 origin-containing plasmid topoisomer distribution. The loss of up to...

Tripathy, S.; Berger, E.J., 2012: Quasi-linear viscoelastic properties of costal cartilage using atomic force microscopy. Costal cartilage (CC) is one of the load-bearing tissues of the rib cage. Literature on material characterisation of the CC is limited. Atomic force microscopy (AFM) has been extremely successful in characterising the elastic properties of soft bi...

Sweeney, B., 1985: Nitrous oxide: panacea or poison?. Saad Digest 6(4): 82-88

Hughes, Barry., 1994: George Soper Cansdale - the 'zoo man'. Nigerian Field. April; 591-2: 3-6

Frishman, W.H.; Charlap, S., 1984: Nifedipine in the treatment of systemic hypertension. Archives of Internal Medicine 144(12): 2335-2336

Hartert, E., 1916: One of the rarest Birds (Callaeops periopthalmica). Novitates Zoologicae, 23 335-336

Bose, S.K., 1995: Sneddon-wilkinson disease and arthritis. Sneddon-Wilkinson disease (subcorneal pustular dermatosis) is an uncommon disorder. An unusual association with seronegative arthritis is reported with review of literature.

Mahmood, A.; Alvarado, F., 1975: The activation of intestinal brush border sucrase by alkali metal ions: an allosteric mechanism similar to that for the Na+-activation of nonelectrolyte transport systems in intestine. Archives of Biochemistry and Biophysics 168(2): 585-593

Yasojima, K.; Tsujimura, A.; Mizuno, T.; Shigeyoshi, Y.; Inazawa, J.; Kikuno, R.; Kuma, K.I.hi; Ohkubo, K.; Hosokawa, Y.; Ibata, Y.; Abe, T.; Miyata, T.; Matsubara, K.; Nakajima, K.; Hashimoto Gotoh, T., 1997: Cloning of human and mouse cDNAs encoding novel zinc finger proteins expressed in cerebellum and hippocampus. We identified a novel gene, kf-1, highly expressed in the normal cerebellum but not in the cerebral cortex, the expression of which could have been augmented in the cerebral cortex of a sporadic Alzheimer's disease patient. We cloned human an...